The treatment produced some of the same benefits as eating fewer calories and appeared to improve several measures beyond what calorie restriction alone could explain.
A small laboratory study found that four popular smartwatches often missed the mark on calories burned during cycling, with errors tending to grow as body fat increased.
All three diets improved metabolic health after similar weight loss, but keto led to larger short-term improvements in liver fat and some blood sugar measures.
In focus groups with 24 UK men, dieting was often described as restrictive and female-focused, while simple goals, accountability and personal challenges sounded more appealing.
Unlike medications that help people eat less, the experimental compound increased how much energy mice used. It has not yet been tested in humans.
A large international study finds that anorexia nervosa and binge eating have genetic links that cannot simply be explained by a person’s weight.
Young people who moved from overweight or obesity into a normal BMI range were less likely to progress to clinical type 1 diabetes, though the study cannot show that changing weight caused the difference.
In a small six-month trial, people who limited eating to noon through 8 p.m. improved a key measure of blood sugar control, though they did not lose significantly more weight.
Communities with more dollar stores had higher rates of obesity, high blood pressure and type 2 diabetes, but the study cannot show the stores caused those outcomes.
Personalized dietitian feedback kept users tracking meals, while peer comparisons sometimes pushed less healthy eaters away.
People who kept eating pork during a six-month WeightWatchers program still lost meaningful weight and improved diet quality, but those who avoided pork lost more.
A study suggests weight lost before diagnosis may make higher BMI appear protective.
Among bariatric surgery candidates with similar BMIs, those classified with clinical obesity had more health problems and higher estimated risks.
Patients taking GLP-1 medications before bariatric surgery lost about the same amount of weight one year later as those who were not taking the drugs.
When researchers disrupted a protein in appetite-regulating brain cells, mice ate more fat and gained more weight, offering clues about how the brain helps control eating.
A study in female mice found that brain cells known for driving hunger were unexpectedly needed for the full weight-lowering effects of GLP-1 drugs.
Adding nutrition, exercise and behavioral support to ongoing obesity care did not significantly increase the chances of losing at least 5% of body weight, though some metabolic measures improved.
A new review finds that obesity and aging affect many of the same biological systems, but shared pathways do not mean obesity literally makes someone older or that weight loss can turn back the clock.
Adults ate about the same weight of food after adding more vegetables, fruit and beans, but the analysis cannot prove lower energy density caused their weight loss.
Experts advise against using GLP-1 medications during pregnancy or breastfeeding while offering guidance on birth control, pregnancy planning and accidental exposure.
A large randomized trial found that adding structured family treatment to enhanced pediatric care improved children’s weight trajectories, quality of life and household routines.
Four photo-based tracking tools underestimated calories and fat in a preliminary study of carefully prepared meals, suggesting their automatic estimates should be treated as a starting point rather than a precise measurement.
A mouse study offers a possible explanation for why activating or blocking the same appetite-related receptor can both support weight loss, but it does not show which approach will work best in people.
A new expert perspective questions whether evidence from weight-management research supports recommending 1.2 to 1.6 grams of protein per kilogram to adults across the lifespan.
A large observational study found lower hospitalization rates among adults with alcohol-use disorder who started semaglutide or tirzepatide, but it cannot show that the drugs reduced drinking or treated alcohol dependence.
